๐๐ค๐ถ๐ต๐ฆ ๐ฑ๐ฐ๐ด๐ต๐ฆ๐ณ๐ช๐ฐ๐ณ ๐ฎ๐ถ๐ญ๐ต๐ช๐ง๐ฐ๐ค๐ข๐ญ ๐ฑ๐ญ๐ข๐ค๐ฐ๐ช๐ฅ ๐ฑ๐ช๐จ๐ฎ๐ฆ๐ฏ๐ต ๐ฆ๐ฑ๐ช๐ต๐ฉ๐ฆ๐ญ๐ช๐ฐ๐ฑ๐ข๐ต๐ฉ๐บ (๐๐๐๐๐๐)โฃ
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โ White Dot Syndromeโฃ
โ Inflammatory chorioretinopathy which was first described by Gass in 1968.โฃ
โ Bilateral, M=F, 20-40 yearsโฃ
โ Association with HLAB7 and HLADR2,โฃ
โ Could be an immune driven vascular alterationโฃ
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๐๐ฐ๐ฏ๐ฅ๐ช๐ต๐ช๐ฐ๐ฏ๐ด ๐ข๐ด๐ด๐ฐ๐ค๐ช๐ข๐ต๐ฆ๐ฅ ๐ธ๐ช๐ต๐ฉ ๐๐๐๐๐๐ โฃ
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๐๐ฏ๐ง๐ฆ๐ค๐ต๐ช๐ฐ๐ถ๐ดโฃ
โ Group A streptococcalโฃ
โ Adenovirus type 5โฃ
โ Tuberculosisโฃ
โ Lyme diseaseโฃ
โ Mumpsโฃ
โ Hepatitis B vaccinationโฃ
โฃ
๐๐ฐ๐ฏ๐ช๐ฏ๐ง๐ฆ๐ค๐ต๐ช๐ฐ๐ถ๐ดโฃ
โ Erythema nodosumโฃ
โ Wegener granulomatosisโฃ
โ PANโฃ
โ Cerebral vasculitisโฃ
โ Scleritisโฃ
โ Episcleritisโฃ
โ Sarcoidosisโฃ
โ Ulcerative colitisโฃ
โฃ
๐ฆ๐๐บ๐ฝ๐๐ผ๐บ๐:โฃ
โ Preceding viral or flu like illnessโฃ
โ Rapid onset of blurred vision bilaterally, and asymmetricโฃ
โ Fellow eye involved within days to weeksโฃ
โ Central and paracentral scotomasโฃ
โ Photopsias before vision lossโฃ
โ Headache and neurological symptomsโฃ
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๐ฆ๐ถ๐ด๐ป๐:โฃ
โ VA 20/40- CFโฃ
โ Mild anterior uveitisโฃ
โ Mild to moderate vitritis 50%โฃ
โ ๐๐ช๐ฃ๐๐ช๐จ - ๐ข๐ช๐ก๐ฉ๐๐ฅ๐ก๐ ๐๐ก๐๐ฉ ๐๐๐ก๐๐ฉ๐๐ง๐๐ก ๐ฎ๐๐ก๐ก๐ค๐ฌ-๐ฌ๐๐๐ฉ๐ ๐ฅ๐ก๐๐๐ค๐๐ ๐ก๐๐จ๐๐ค๐ฃ๐จ ๐๐ฉ ๐ก๐๐ซ๐๐ก ๐ค๐ ๐๐๐ ๐๐ฃ๐ ๐๐๐ค๐ง๐ค๐๐ ๐ท-๐ธ ๐๐๐จ๐ ๐๐๐๐ข๐๐ฉ๐๐ง๐จ ๐ฅ๐ค๐จ๐ฉ๐๐ง๐๐ค๐ง ๐ฉ๐ค ๐๐ฆ๐ช๐๐ฉ๐ค๐งโฃ
โ New peripheral lesions can appear in a linear or radial array over the next 3 weeksโฃ
โ Papillitis seen, CME uncommonโฃ
โ Atypical findings- retinal vasculitis, RVOs, retinal neovascularization, exudative RDsโฃ
โ Fade over 1-2 weeks, resolves over 2-6 weeksโฃ
โ Older lesions --- well defined alteration in RPE consisting of alternating areas of depigmentation and hyperpigmentation due to pigment clumpingโฃ
โ Variant - relentless placoid chorioretinitis - has features of APMPPE and serpiginous choroiditisโฃ
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Investigations and Management to follow soon. โฃ
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www.ophthalmobytes.comโฃ
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